Search results for "Complement C3b Inactivator Proteins"

showing 5 items of 5 documents

Activation of the alternative pathway of complement: efficient fluid-phase amplification by blockade of the regulatory complement protein β1H through…

1981

Current concepts of activation of the alternative pathway of complement (APC) focus on the central role of an amplification mechanism triggered by C3b which is covalently bound to the surfact of activating substances. Using sulfated polyanions as model substances, an efficient fluid-phase activation of complement is demonstrated in contrast to solid-phase activation. It is shown that particulate high-molecular weight sulfated polyanions are capable of reversible binding the guinea pig and human regulatory protein beta1H. This fixation leads to an extensive activation of C3 and factor B because the regulatory function of beta1H is blocked in the fluid-phase C3b-dependent amplification system…

AnionsChemical PhenomenaComplement Pathway AlternativeGuinea PigsImmunologyBiologyComplement factor BAbsorptionGuinea pigSulfationComplement C3b Inactivator ProteinsAnimalsHumansImmunology and AllergyComplement ActivationRegulation of gene expressionChemistry PhysicalSulfatesGoatsImmune SeraComplement C3Complement systemCell biologyKineticsBiochemistryCovalent bondComplement Factor HComplement C3bAlternative complement pathwayFunction (biology)European Journal of Immunology
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Cyclic AMP-mediated upregulation of the expression of neuronal NO synthase in human A673 neuroepithelioma cells results in a decrease in the level of…

2004

The expression level of neuronal nitric oxide synthase (nNOS) can vary depending on the (patho)physiological conditions. Here we document a marked induction of nNOS mRNA, protein, and total NO production in response to dibutyryl cyclic AMP (db-cAMP) in human A673 neuroepithelial cells. However, the upregulation of nNOS was associated with a decreased level of production of bioactive NO and by an increase in the level of generation of reactive oxygen species (ROS). ROS production could be prevented by the NOS inhibitor L-NAME, suggesting nNOS itself is involved in ROS generation. Sepiapterin supplementation of db-cAMP-treated A673 cells could restore full bioactive NO production, most likely…

CAMP-Responsive Element ModulatorNitric Oxide Synthase Type IBiologyCREBNitric OxideBiochemistryAdenylyl cyclaseCyclic AMP Response Element Modulatorchemistry.chemical_compoundMiceNeuroblastomaCoactivatorComplement C3b Inactivator ProteinsCyclic AMPAnimalsHumansNeuroectodermal Tumors Primitive PeripheralCREB-binding proteinEnzyme InhibitorsProtein kinase AeducationCyclic AMP Response Element-Binding ProteinGTP CyclohydrolaseCAMP response element bindingHomeodomain ProteinsNeuronseducation.field_of_studyForskolinPhosphoric Diester HydrolasesIntracellular Signaling Peptides and ProteinsBlood ProteinsLIM Domain ProteinsMolecular biologyCyclic AMP-Dependent Protein KinasesPterinsUp-RegulationDNA-Binding ProteinsRepressor ProteinsAntisense Elements (Genetics)NG-Nitroarginine Methyl EsterchemistryBucladesineGene Expression RegulationComplement Factor Hbiology.proteinNitric Oxide SynthaseReactive Oxygen SpeciesSignal TransductionBiochemistry
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Importance of Factors H and I for the Adherence of C3b-Coated Erythrocytes to Cells

1983

Abstract The role of cell membrane-associated human factor H for the binding of cell-bound Cab to complement receptor-carrying (CR + ) cells was investigated. Pretreatment of CR + cells with antibodies to factor H inhibited the adherence of Cab-coated red cells to human tonsil lymphocytes (TL) and peripheral blood monocytes (Mo). The Cab receptor reactivity of human polymorphonuclear leucocytes (PMN) was not influenced and the one of Raji lymphoblastoid cells only slightly influenced; iC3b and Cad receptor reactivity was in no case affected. When diisopropylfluorophosphate (DFP) in a concentration of 0.1 mM was present during pretreatment of the CR + cells with anti H, the antibodies gained…

ErythrocytesIsoflurophateRosette Formationmedicine.drug_classLymphocyteComplement Pathway AlternativeImmunologyMonoclonal antibodyMonocytesImmunoglobulin Fab FragmentsComplement C3b Inactivator ProteinsmedicineAnimalsHumansImmunology and AllergyLymphocytesComplement ActivationbiologyChemistryLymphoblastfungifood and beveragesHematologyMolecular biologyReceptors ComplementComplement systemRaji cellmedicine.anatomical_structureBiochemistryComplement Factor HFactor HReceptors Complement 3bbiology.proteiniC3bRabbitsAntibodyImmunobiology
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Role of β1H for the binding of C3b-coated particles to human lymphoid and phagocytic cells

1981

Coating of EAC14oxy23b with highly purified human serum beta 1H globulin (beta 1H) led to acceleration of rosette formation with human peripheral blood lymphocytes (PBL), tonsil lymphocytes, B lymphoblastoid (Raji) cells, granulocytes and monocytes. This reaction was discernible from C3bi-dependent rosette formation. Enhancement of rosette formation of C3b cells by beta 1H was most effective at limiting amounts of C3 per EAC14oxy23b. The beta 1H effect was not due to trace contamination with C3b inactivator. beta 1H-dependent rosette formation with the various lymphoid and phagocytic cells could be suppressed by the F(ab')2 fragment of anti-beta 1H suggesting beta 1H-mediated binding of bet…

ErythrocytesRosette FormationGlobulinGuinea PigsImmunologyTurn (biochemistry)Immunoglobulin Fab FragmentsComplement C3b Inactivator ProteinsmedicineAnimalsHumansImmunology and AllergyLymphocytesBeta (finance)ReceptorPhagocytesBinding SitesSheepbiologyGoatsLymphoblastMolecular biologyReceptors ComplementRaji cellmedicine.anatomical_structureRosette formationComplement Factor HTonsilComplement C3bImmunologybiology.proteinEuropean Journal of Immunology
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Combined homozygous factor H and heterozygous C2 deficiency in an Italian family

1988

Three of four children in a family have homozygous (less than 1% of normal) deficiency of factor H of the complement system and both parents, who are first cousins, are heterozygous for the same defect. The father and two of the H-deficient siblings also have a partial C2 deficiency. One of the children with combined deficiencies is affected by systemic lupus erythematosus with nephritis. No increased susceptibility to infections has been observed in the family. H deficiency is inherited in an autosomal codominant manner and is independently transmitted from C2 deficiency and HLA haplotypes. In the homozygous state it is associated with very low serum concentrations of B and C3, barely demo…

Heterozygotemedicine.medical_specialtyGenetic LinkageImmunologyHLA AntigensInternal medicineComplement C3b Inactivator ProteinsmedicineHumansLupus Erythematosus SystemicImmunology and AllergyChildImmunoelectrophoresisLupus erythematosusComplement component 2business.industryHomozygoteHeterozygote advantageComplement C2Complement deficiencymedicine.diseasePedigreeComplement systemEndocrinologyComplement Factor HFactor HComplement C3bImmunologyProperdinFemalebusinessNephritisComplement Factor BJournal of Clinical Immunology
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